Relationships of occupational and non-occupational physical activity to abdominal obesity: Author Interview

Relationships of occupational and non-occupational physical activity to abdominal obesity

Author Interview : Jeremy Steeves Ph.D., MPH candidate 2012
Bloomberg School of Public Health
Johns Hopkins University
624 N Broadway Baltimore, MD 21205

What are the main findings of the study?

We found that highly active jobs may protect against the risk of
abdominal adiposity (a waist circumference > 88 cm in women and > 102
cm in men), even in the absence of physical activity outside of work.
This study utilized data from the 1999-2006 National Health and
Nutrition Examination Survey, and assessed the association between
total leisure-time physical activity (leisure-time, transport and
domestic) and occupational activity with abdominal obesity in 3890
working adults.

We found that the total volume of physical activity, both at work and
outside of work, is linked to adiposity. For sedentary employees who
do not engage in physical activity outside of work, the likelihood of
having abdominal obesity may be reduced by about half if they were
employed in an occupation that involved high levels of activity
(cleaning and building service occupations, construction trades, farm
and nursery workers, laborers, waiters and waitresses etc).

Were any of the findings unexpected?

While, the Physical Activity Guidelines for Americans highlight that
the total volume of physical activity has an impact on health, many
may still believe that the benefits of physical activity are
restricted to “exercisers.”

Despite the reduction of occupations that
require manual labor, our results suggest that some occupations still
provide enough physical activity to reduce the likelihood of abdominal
obesity.

What should clinicians and patients take away from this study?

Everyone should be encouraged to engage in regular physical activity
either through work or play. Habitual physical activity acquired in
any form adds up, and is important for long-term weight management.
Leisure-time, occupational, household activity, and active commuting
all count towards total physical activity and health promotion
(lowering all-cause mortality, lowering cardiovascular mortality,
improving function and enhancing quality of life) .

For individuals who work in sedentary jobs- which is most of us,
myself included- there is an increased emphasis on the need to acquire
physical activity throughout the day, and outside of work in order to
reduce the risk of abdominal adiposity.

What recommendations do you have for future research as a result of your study?

The addition of occupational physical activity into the measurement of
total physical activity is still in its infancy. As both occupational
activity and leisure-time physical activity are inversely related to
abdominal obesity, future research should focus on doing a better job
measuring occupational activity to fully understand the role of
physical activity in disease prevention. This may also provide a more
accurate estimate of the prevalence of Americans meeting the
recommended levels of physical activity.

Reference:

Relationships of occupational and non-occupational physical activity to abdominal obesity.

Steeves JA, Bassett DR Jr, Thompson DL, Fitzhugh EC.

Department of Kinesiology, Recreation and Sport Studies, The University of Tennessee, Knoxville, TN, USA.
Int J Obes (Lond). 2012 Jan;36(1):100-6. doi: 10.1038/ijo.2011.50. Epub 2011 Mar 22.

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Association of baseline sex hormone levels with baseline and longitudinal changes in waist-to-hip ratio: Dr. Vaidya

Association of baseline sex hormone levels with baseline and longitudinal changes in waist-to-hip ratio: Multi-Ethnic Study of Atherosclerosis

Author Interview: Dhananjay Vaidya MD PhD MPH

Assistant Professor of Medicine
Johns Hopkins University

What are the main findings of the study?

In men and postmenopausal women, those with a higher level of naturally occurring estrogen levels tend to become more obese (in terms of waist-to-hip ratio) over time.

Similarly those with a lower level of sex hormone binding protein (SHBG) tend to become more obese over time. Though men with lower testosterone levels are more obese, this is not followed by increasing obesity.

Were any of the findings unexpected?

Our study, along with other studies in the last 5 years, shows that estrogen levels in post-menopausal women are not a health-promoting condition in terms body shape. In past decades, this finding would have been unexpected.

What should clinicians and patients take away from this study?

Because this is an observational study, we should be circumspect about taking away clinical implications. However, it is a strength that we followed up participants over several years: we think that boosting naturally occurring estrogen or testosterone levels in middle aged and older individuals is not likely to improve the time course of obesity.

What recommendations do you have for future research as a result of your study?

To follow up on the previous statement, we need to confirm the effects that treatments to manipulate hormone levels have on obesity. Another area to be researched is about the role of adrenal hormones like DHEA. DHEA can be naturally converted to estrogen or testosterone in the body, but the tendency to be converted to one or the other product differs from person to person. Future research is needed to test if conversion of DHEA to androgens or estrogens is associated with increasing obesity.

Reference:

Association of baseline sex hormone levels with baseline and longitudinal changes in waist-to-hip ratio: Multi-Ethnic Study of Atherosclerosis

D Vaidya, A Dobs, S M Gapstur, S H Golden, M Cushman, K Liu and P Ouyang
International Journal of Obesity , (24 January 2012) | doi:10.1038/ijo.2012.3

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Circulating ACE is a predictor of weight loss maintenance not only in overweight and obese women, but also in men : Authors’ Interview

Circulating ACE is a predictor of weight loss maintenance not only in overweight and obese women, but also in men

Author interview: Edwin C.M. Mariman and Ping Wang 

Edwin C.M. Mariman, PhD,
Professor of Functional Genetics,
NUTRIM School for Nutrition, Toxicology and Metabolism
Maastricht University Medical Centre + (MUMC+)
Maastricht, The Netherlands.

What are the main findings of the study?

In a group of obese men it was demonstrated that the degree, by which the ACE concentration in blood changes during weight loss, is a predictor of the risk for weight regain during a follow-up period of 6 months. It confirms recent finding in females (Wang et al., PLoS One 2011 6(2): e16773), in whom the predicting power of ACE seems even stronger. ACE is now one of very few physiological predictors of weight regain after weight loss.

Were any of the findings unexpected?

ACE is well known for regulating blood pressure and a relation with weight regain or weight maintenance was quite unexpected, although the gene for ACE has been found associated with obesity.

What should clinicians and patients take away from this study?

ACE might become part of a diagnostic tool identifying subjects with increased risk for weight regain after weight loss, who can then be given extra guidance to prevent weight regain.

What recommendations do you have for future research as a result of your study?

Many questions remain to be answered. The biology behind the relation between ACE and weight regain/maintenance is presently unknown. But a clear message from this research seems to be that such metabolic predictors can be found and this warrants a more intensive search to find them. It will learn us more about the mechanisms behind weight regain and may be helpful to sustain long term weight maintenance by using those predictors to identify subjects who are at high risk for weight regain. However, at this moment this is all still in a preliminary stage.

Reference:

Circulating ACE is a predictor of weight loss maintenance not only in overweight and obese women, but also in men

P Wang, C Holst, W K W H Wodzig, M R Andersen, A Astrup, M A van Baak, T M Larsen, S A Jebb, A Kafatos, A F H Pfeiffer, J A Martinez, T Handjieva-Darlenska, M Kunesova, N Viguerie, D Langin, W H M Saris, E C M Mariman and on behalf of Diogenes consortium

International Journal of Obesity , (24 January 2012) | doi:10.1038/ijo.2011.278

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History matters: childhood weight trajectories as a basis for planning community-based obesity prevention to adolescents : Author Interview

History matters: childhood weight trajectories as a basis for planning community-based obesity prevention to adolescents

Author Interview: Joakim Ekberg MSc
Department of Medical and Health Sciences
Faculty of Health Sciences
Linköping University
SE-581 83 Linköping Sweden

What are the main findings of the study?

Using projections from epidemiological data and regional practice, we
found that by basing the selection of adolescents for obesity
prevention on weight trajectories, the load on highly specialized
paediatric care can be reduced by one–third and total health service
costs for obesity management among adolescents reduced by one–third.

However, this was a hypothetical comparison and before used in
policies and prevention program planning, the findings warrant
confirmation in prospective cost–benefit studies.

Were any of the findings unexpected?

The purpose of the analysis was to examine tailoring treatments to the
needs of the patients on a population level based on readily available
epidemiological data.

We did however not expect the proposed suggestion to reduce the total health service costs to the extent that it did.

What should clinicians and patients take away from this study?

In light of that treatment of all obese adolescents often is not
fiscally feasible, usually a higher BMI cut-off is used to determine
which obese adolescents to treat, e.g. BMI>35.
The article instead suggests that this decision could be explicitly based on weight
trajectory and treatment history. Nothing can replace clinical
experience and shared decisions with the patient in the individual
case, however having simple heuristics based on readily available data
may aid in screening.

What recommendations do you have for future research as a result of your study?

We would very much like to see a prospective cost-benefit study using
the suggested trajectory based program.

Reference:

History matters: childhood weight trajectories as a basis for planning community-based obesity prevention to adolescents

J Ekberg, M Angbratt, L Valter, M Nordvall and T Timpka
International Journal of Obesity , (17 January 2012) | doi:10.1038/ijo.2011.263

 

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Brown adipose tissue oxidative metabolism contributes to energy expenditure during acute cold exposure in humans : Dr. Carpentier

Brown adipose tissue oxidative metabolism contributes to energy expenditure during acute cold exposure in humans

Author Interview: André Carpentier MD FRCPC

Professor Department of Medicine
CIHR-GSK Chair in Diabetes
Centre de recherche clinique Etienne-Le Bel
Université de Sherbrooke

What are the main findings of the study?

The main findings are that healthy men when exposed to cold can significantly increase their caloric expenditure through activation of ‘fat burning’ in the brown fat.

We designed our experiments to minimize shivering in our participants so that energy expenditure from this source is minimized. We quantified brown fat glucose and fat uptake from circulation during the experiments and found that a very small part of the 250 extra kilocalories burned during the 3-hour cold exposure was accounted by these sources. However, there was a significant reduction of the fat stored inside brown fat during the experiment. Furthermore, the more brown fat an individual has, the less the need to shiver to expand the same amount of calories.

Were any of the finding unexpected? 

Unexpected? I was personally sceptical that such a small tissue (brown fat is about 1 to 3% of the total amount of fat in the body) can significantly contribute to energy expenditure. I became convinced by our experimental data. This was a very controversial and unproven issue in humans, although we had solid evidence in small animals thatbrown adipose tissue contribution to energy expenditure can be very important.

What should clinicians and patients take home from your study?

Take home: Please don’t throw yourself naked in the snow and don’t buy expensive cooling devices in the hope to lose weight!

Much more study is needed to determine whether this mechanism can be safely maintained over time and successfully used to tip the balance away from chronic caloric excess. We need to determine how to accomplish this safely in humans.

We also need to find out the potential long-term adaptations of the body in that instance (i.e. whether there would be compensatory increase in energy intake or other counter adaptation of the body to prevent weight loss in that instance).

Ours is a proof-of-concept study. Now we have a potential target to work on. It is only the start. Turning down the heat in your apartment is good to the environment and for your wallet. However, it is premature to claim it would be good for your waist as well.

What  further research are you planning as a result of this study?

This study is funded by the Canadian Diabetes Association. With my colleagues Dr Denis Richard from Laval University, François Haman from University of Ottawa, and Eric Turcotte from University of Sherbrooke, we will now repeat these experiments in subjects with diabetes to determine whether their brown fat display abnormal metabolism and fat burning capacity.

We already know that people with diabetes have less spontaneously active brown fat when assessed by looking at glucose uptake in that tissue. We don’t known whether this means that all brown fat metabolism is lower in these individuals. We don’t know the mechanism of this finding either. We are setting up to do these experiments.

Reference:

Brown adipose tissue oxidative metabolism contributes to energy expenditure during acute cold exposure in humans

Véronique Ouellet, Sébastien M. Labbé, Denis P. Blondin, Serge Phoenix, Brigitte Guérin, François Haman, Eric E. Turcotte, Denis Richard, André C. Carpentier
J Clin Invest. 2012; doi:10.1172/JCI60433

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Competitive food sales in schools and childhood obesity: Authors’ Interview

Competitive food sales in schools and childhood obesity: A longitudinal study. 

Author Interview:  Jennifer Van Hook and Claire E. Altman

Jennifer Van Hook
Director, Population Research Institute
Professor of Sociology & Demography
601 Oswald Tower
University Park, PA 16802
814-867-2276

What are the main findings of the study?

We find that weight gain has nothing to do with the candy, soda, chips, and other junk food middle school students can purchase at school.  The research relies on data from the Early Childhood Longitudinal Study, Kindergarten Class of 1998-1999, which follows a nationally representative sample of students from the fall of kindergarten through the spring of eighth grade. We used a subsample of 19,450 children who attended school in the same county in both fifth and eighth grades.

We found that 59.2 percent of fifth graders and 86.3 percent of eighth graders in our study attended schools that sold junk food. But, while there was a significant increase in the percentage of students who attended schools that sold junk food between fifth and eighth grades, there was no rise in the percentage of students who were overweight or obese. In fact, despite the increased availability of junk food, the percentage of students who were overweight or obese actually decreased slightly from fifth grade to eighth grade.

Were any of the findings unexpected?

Yes, our findings seem to defy common sense.  After all, we all know that children who eat junk food and soda are more likely to gain weight.  But to understand what is going on, we need to take into consideration all of the places where children eat. Schools only represent a small portion of children’s food environment. Children can get food at home and their neighborhoods, and many children can walk down the street from the school to buy food. Additionally, children are actually very busy at school, so there isn’t much opportunity for them to eat while they’re in school, or at least snack endlessly, compared to when they’re at home.  School days are scheduled from beginning to end, including time spent eating. This differs considerably from home environments, where mealtimes are less regular, eating blends with other activities such as TV viewing, opportunities for snacking are greater, and food consumption is less closely monitored, especially for children staying home alone.

What should clinicians and parents take away from this study?

Obesity is a significant health issue facing children today. The percentage of obese children tripled between the early 1970s to the late 2000s. Diet and physical activity play an important role in obesity. However, childhood obesity cannot be placed solely in the hands of a single institution such as schools.  Instead, to combat childhood obesity a coordinated response among educators, health care workers, parents, businesses, and government is probably required.  This message may come as a disappointment to those hoping for an easy solution.

Additionally, this study suggests that we need to think more holistically about children’s’ food environments. Food is ubiquitous so the elimination of junk food sales in schools is unlikely to make much of a dent in the child obesity epidemic.

What recommendations do you have for future research as a result of your study?

Future research needs to take into account children’s development. Children’s food preferences and eating habits are firmly established early in life, so middle school environment may not matter much.  Early childhood experiences and home environments have profound effects on children’s dietary patterns.  For example, some research suggests that children can lose the ability to self-regulate food consumption (and stop eating when full) in early childhood, largely as a consequence of child feeding practices. This suggests that when it comes to combating childhood obesity and weight issues, researchers should put more emphasis on younger children.

Reference:

Competitive Food Sales in Schools and Childhood Obesity: A Longitudinal StudyJennifer Van Hook and Claire E. Altman

Sociology of Education, January 2012;
vol. 85, 1: pp. 23-39.,  
first published on August 8, 2011

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Association of Variations in TPH1 & HTR2B with Gestational Weight Gain & Obesity: Dr. Park

Association of Variations in TPH1 and HTR2B with Gestational Weight Gain and Measures of Obesity.

Author Interview: Kyong Soo Park, M.D., Ph.D.

Professor, Department of Internal Medicine
WCU Department of Molecular Medicine and Biopharmaceutical Sciences,
Seoul National University College of Medicine

What are the main findings of the study?

We found that genetic variants in tryptophan hydroxylase 1 (TPH1), the rate limiting enzyme in the synthesis of serotonin were associated with decreased weight gain during pregnancy in gestational diabetes mellitus (GDM) women and increased waist circumference and body mass index (BMI) in the general population. However, variants in TPH1 were not associated with risk of GDM.

Were any of the findings unexpected?

Serotonin is well known for its role in regulating appetite, mood, and body weight. It has been recently reported that serotonin is also very important in pancreatic beta cell proliferation during pregnancy.

Specifically, genetic alteration in TPH1 or serotonin receptor 2b (HTR2B) resulted in glucose intolerance in mouse. The primary objective of our study was to evaluate the genetic association of TPH1 and HTR2B with risk of GDM in humans.

Unfortunately, there was no significant association between genetic variants in these two genes and risk of GDM. However, we found that TPH1 variants were associated with decreased weight gain during pregnancy. In addition, the TPH1 variants were also associated with waist circumference and BMI in a population based cohort comprised of 8842 subjects.

The opposite direction of association of TPH1 variants, that is decreased weight gain during pregnancy and increased body mass in general population, first seemed contradictory.
However, these might reflect the fact that obese women tend to gain less weight during pregnancy.

What should clinicians and patients take away from this study?

We found that genetic variants in serotonin system are associated with body mass regulation.
These findings are in line with the fact that drugs modulating serotonin system, such as fenfluramine-phentermin and locaserine, result in satiety and weight loss. However, further research is required for the translation of our findings into clinical practice.

What recommendations do you have for future research as a result of your study?

The association of TPH1 variants and measures of obesity requires further replication study in a different population for confirmation. In addition, it would be interesting to elucidate the functional role of TPH1 in energy intake and expenditure in humans.

The serotonin system including its various receptors is an active area of research for anti-obesity drug development.

We hope in depth research in TPH1 and serotonin receptors will lead us to better understanding of the mechanism of body weight regulation and development of novel therapeutic strategies

Reference:

Association of Variations in TPH1 and HTR2B with Gestational Weight Gain and Measures of Obesity.

Kwak SH, Park BL, Kim H, German MS, Go MJ, Jung HS, Koo BK, Cho YM, Choi SH, Cho YS, Shin HD, Jang HC, Park KS.
Department of Internal Medicine, Seoul National University College of Medicine,
Seoul, Korea.
Obesity (Silver Spring). 2012 Jan;20(1):233-8. doi: 10.1038/oby.2011.253.

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Acute Sleep Deprivation Enhances the Brain’s Response to Hedonic Food Stimuli : Dr. Benedict

Acute Sleep Deprivation Enhances the Brain’s Response to Hedonic Food Stimuli: An fMRI Study

Author Interview: Christian Benedict
Department of Neuroscience, Uppsala University
Box 593, SE-751 24 Uppsala, Sweden

What are the main findings of the study?

Sleep-deprived students showed an increased activation in a brain region related to appetite motivation while they viewed images of food the next morning.

This increased brain activation was related to their appetite sensation in that they rated the foods as more appetizing than they did after one night of sleep.

Were any of the findings unexpected?
No.

What should clinicians and patients take away from this study?

That sleep deprivation may contribute to weight gain in the long run by making foods more appetizing.

What recommendations do you have for future research as a result of your study?

Bearing in mind that our volunteers had no chance to sleep at all, our findings should be confirmed under conditions of partial sleep deprivation.

Reference:
Acute Sleep Deprivation Enhances the Brain’s Response to Hedonic Food Stimuli: An fMRI Study
Christian Benedict,  Samantha J. Brooks, Owen G. O’Daly, Markus S. Almèn, Arvid Morell,Karin Åberg, Malin Gingnell, Bernd Schultes, Manfred Hallschmid, Jan-Erik Broman, Elna-Marie Larsson, and Helgi B. Schiöth
JCEM jc.2011-2759; doi:10.1210/jc.2011-2759

 

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Study: Pictures of food create feelings of hunger

Max Planck researchers have proven something scientifically for the first time that laypeople have always known: the mere sight of delicious food stimulates the appetite. A study on healthy young men has documented that the amount of the neurosecretory protein hormone ghrelin in the blood increases as a result of visual stimulation through images of food. As a main regulator, ghrelin controls both eating behaviour and the physical processes involved in food metabolism. These results show that, in addition to the physiological mechanisms for maintaining the body’s energy status, environmental factors also have a specific influence on food consumption. Thus, the pervasive presence of appetising food in the media could contribute to weight increase in Western populations.

Warning: “Avoid looking at pictures of appetising food as it will make you hungry!” Dieticians could be making recommendations along these lines in the future. It has long been known that, in addition to the physiological regulatory circuits for the maintenance of a sufficient energy status for the body, external stimuli like smell or the sight of food also influence our feelings of hunger and our resulting eating behaviour. The danger that the exposure to such images will result in the consumption of food that is not needed to maintain the body’s energy status is particularly high in our advertising-dominated society.

In a study involving healthy male subjects, Axel Steiger and his research group at the Max Planck Institute of Psychiatry investigated the molecular processes for the control of food consumption. They examined the specific physiological reaction of the test subjects to images showing either delicious food or non-edible objects. The concentrations of different hormones in the blood such as grehlin, leptin and insulin, which play a role in the regulation of food consumption, were measured. The researchers actually observed that the concentration of grehlin in the blood increases specifically in response to visual stimulation with food images.

“The findings of our study demonstrate, for the first time, that the release of ghrelin into the blood for the regulation of food consumption is also controlled by external factors. Our brain thereby processes these visual stimuli, and the physical processes that control our perception of appetite are triggered involuntarily. This mechanism could prompt us to eat a piece of cake just two hours after breakfast,” says Petra Schüssler, a scientist at the Max Planck Institute. She thus recommends that individuals with weight problems should preferably avoid looking at images of appetising food.

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Combating childhood obesity: a survey of laws affecting the built environments of low-income and minority children : R. Lindholm

Combating childhood obesity: a survey of laws affecting the built environments of low-income and minority children.

Author Interview: Raymond Lindholm
Georgia State University College of Law, Center for Health, Law, and Society
Atlanta, GA 30303-3047, USA

What are the main findings of the study?

My research focused on state responses to the dramatic rise in childhood obesity, especially with regard to the built environments of low-income and minority children.  Specifically, I was interested in what the scientific literature had to say regarding the relationship between the built environment and childhood obesity, and what types of legal strategies State’s are using to regulate the built environment to provide greater access to healthy food sources, decrease access to unhealthy food sources, and provide more physical activity resources for the most affected children.

What I found is that there is growing scientific evidence to support the connection between a child’s built environment and his or her risk of being overweight or obese.  Specifically, many studies indicate that low-income and minority children are more likely to live in neighborhoods characterized by higher crime rates, more abandoned and dilapidated homes, more trash and debris, more traffic and congestion, much more fast-food restaurants and convenience stores, less access to grocery stores, less access to green spaces, less access to physical activity resources, and less access to sidewalks and neighborhood connectivity.  Children who live in environments characterized by these physical attributes are much more likely to be racial minorities and/or low-income.  They are also much more likely to be overweight or obese than children whose physical environments don’t share these characteristics.

There are a number of ways that States and localities are attempting to regulate these built environments in order to provide greater access to healthy foods, decrease access to unhealthy foods, and increase access to green space and physical activity resources.  Some of these include creating task forces to reduce red-tape, expedite permitting, and provide low-cost financing for large grocery stores wanting to enter low-income markets.  The Pennsylvania Fresh Food Financing commission is a prime example of this type of initiative.  States are also passing laws promoting “Farm-to-School” programs where school children learn about growing food, cultivate school gardens, and learn how to create meals using the produce they grow.

Cities are also establishing initiatives for community gardens, and States are passing laws ensuring protections for communities that wish to have these gardens.  States are using zoning regulations to decrease the number and density of fast-food restaurants in low-income areas.  Los Angeles, California, for example, passed a moratorium on the construction of any new fast-food restaurants in certain areas of Los Angeles characterized by high fast-food density and low property values.  Oakland, California, recently made the news for passing regulations banning “Happy Meals” that offer children toys along with certain fast-food items.

States are increasing access to physical activity resources in several innovative ways.

  • First, many States are passing laws allowing, and even encouraging, joint-use agreements which allow communities to enter into agreements with local schools so that community members may use school facilities and equipment before and after school hours and on weekends.  Some school districts are even using these agreements to help share the cost of developing new facilities with the local communities which will benefit from their use.  This is a simple and cost-effective way to create more access to physical activity resources in low-income areas.
  •  Second, States, with the help of Federal money, are also encouraging children to walk or bike to school through initiatives such as the Safe Routes to School Program.  Forty years ago, the vast majority of children walked or biked to school.  Today, very few children are able to do this.  Safe Routes to School Programs provide money to repair or create physical infrastructure, such as bike paths and sidewalks, so that children can safely walk or bike to school.  These programs may also provide money for cross-walk guards and other monitors to ensure that children reach school safely.
  • Third, States, such as California, are passing “Complete Streets” legislation mandating that new road construction must include bike-lanes and better accommodate alternative forms of transportation, including bikes, pedestrians, and mass transit.

Ultimately, I found that States are using legal tools in a variety of ways to try and combat the childhood obesity epidemic.  Some states, such as Delaware are focusing on education, while states such as Mississippi are mandating that insurers cover bariatric surgery.

Were any of the findings unexpected?

While childhood obesity is increasing in almost every demographic, it was quite alarming to see how disparately low-income and minority children are impacted compared to their white counterparts.  It was also interesting to see the wide spectrum of legal approaches states are using to try and combat childhood obesity.  This speaks to the genius of our federalist system where States can act as laboratories for legal strategies to see what works and what doesn’t.  The fact that many of the states hit hardest by this epidemic, such as Mississippi, haven’t enacted any of these laws was also very interesting.

What should clinicians and patients take away from this study?

For clinicians, I think having a more nuanced understanding of the problem is helpful in and of itself.  Patients and clinicians alike can also actively lobby their state representatives, city officials, and local school boards to try and get some of these initiatives enacted locally. What recommendations do you have for future research as a result of your study?

We still need much more in-depth studies to better understand the relationship between the built environment correlates and obesity.  Also, it is very difficult to quantify the impact that these various laws are having on the problem.  We need much better inter-disciplinary cooperation between scientists, lawyers, and other professionals that can help design, implement, and interpret studies geared toward measuring the impact of such public health laws on public health outcomes in a more precise manner.

Author Interview: Raymond Lindholm

Reference:

Combating childhood obesity: a survey of laws affecting the built environments of low-income and minority children.

Lindholm R.
Georgia State University College of Law, Center for Health, Law, and Society,
Atlanta, GA 30303-3047, USA
Rev Environ Health. 2011;26(3):155-67.

 

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